Adoptive chimeric antigen receptor (CAR) T cell therapy has emerged as an exciting approach for combating cancer. Although CAR T cell therapy has shown remarkable clinical efficacy in hematological malignancies, its success in combating solid tumours has been limited. Here we report that PTPN2-deletion/knock-down/inhibition markedly enhanced the therapeutic efficacy of CAR T cells targeting the human orthologue of murine ErbB2/Neu, HER-2, an oncoprotein which is overexpressed in many solid tumours. PTPN2-deficient CAR T cells significantly repressed tumour growth in mice bearing mammary HER-2 overexpressing tumours which was accompanied by increased tumour T cell infiltration. Moreover, PTPN2-deficiency in HER-2- specific CAR T cells increased CAR T cell homing and activation, whereas PTPN2-inhibition increased the cytotoxic potential of human CAR T cells ex vivo. Our findings define PTPN2 as a target for bolstering T cell mediated anti-tumour immunity and CAR T cell therapy and point towards a novel approach for enhancing the efficacy of adoptive T cell therapy in otherwise recalcitrant solid tumours.